Greg S. Martin, MD
Greg S. Martin, MD
Dr. Martin is associate professor of medicine and associate division director for critical care division of pulmonary, allergy and critical care at Emory University School of Medicine, chief of pulmonary and director of medical and coronary intensive care at Grady Memorial Hospital.
Dr. Martin has been an Emory faculty member since 2000, and chose this specialty because of the opportunity to both help people during their most severe times of medical need, and because of the ability to learn and teach more about this growing field by conducting cutting-edge clinical research. Dr. Martin is clinically active in patient care in both pulmonary and critical care medicine at Grady Memorial Hospital, Emory Midtown Hospital, and Emory University Hospital.
As an active teacher in the Pulmonary and Critical Care fellowship program, Dr. Martin chairs the Division ICU Standardization Committee and serves as the Director of the Medical and Coronary Intensive Care Units at Grady Memorial Hospital and Director of Clinical Core at the Emory Alcohol and Lung Biology Center. In addition, Dr. Martin is Director of the Clinical Interaction Network Site at Emory University Hospital Midtown Hospital and the Atlanta Clinical & Translational Science Institute (ACTSI), serves on the executive committee for the Master of Science in Clinical Research and KL2 - Mentored Clinical and Translational Research Scholars program, ACTSI.
Our critical care research group works on a spectrum of translational studies focusing on two acute and life-threatening critical illnesses: sepsis and acute respiratory distress syndrome (ARDS). Sepsis is the immune response to an infectious stimulus, and ARDS is a form of organ dysfunction that occurs as a consequence of the inflammatory response. Both are unfortunately common and highly lethal. Sepsis is the prototypical condition where acute inflammation is not well organized, compartmentalized or regulated – it is this exaggerated inflammatory response that produces the lethal syndrome known as sepsis. As William Osler described more than 100 years ago, patients with sepsis die because of immune response to the infection, and not from the infection itself. These two entities are linked clinically and biologically with a common pathogenesis stemming from dysregulated oxidative stress and acute inflammation. Our research spans the spectrum from clinical epidemiology and health services research in sepsis and ARDS, to diagnostic and prognostic biomarker studies, through interventional randomized controlled treatment trials.